JURANYI ZSOLT PDF
Jurányi Zsolt is the author of Az alvilág zsoldjában ( avg rating, 16 ratings, 1 review, published ) and Az alvilág csapdájában ( avg rating. nov. Az alvilág csapdájában has 3 ratings and 1 review. Sándor said: Az első rész fényévekkel jobb volt, szerintem. Csak azért vergődtem végig. List of computer science publications by Zsolt Jurányi.
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dblp: Zsolt Jurányi
AB – The most dominant hypotheses for the pathogenesis of schizophrenia have focused primarily upon hyperfunctional dopaminergic and hypofunctional glutamatergic neurotransmission in the central nervous system. Link to publication in Scopus.
Our data indicate that coadministration of an antipsychotic with a GlyT-1 inhibitor may normalize hypofunctional NMDA receptor-mediated glutamatergic neurotransmission with reduced dopaminergic side effects characteristic for antipsychotic medication.
Org injection reduced extracellular dopamine concentrations and elevated extracellular glycine levels but the concentrations of serine and glutamate were not changed. N2 – The most dominant hypotheses jurannyi the pathogenesis of schizophrenia have focused primarily upon hyperfunctional dopaminergic and hypofunctional glutamatergic neurotransmission in the central nervous system.
Neurochemical ResearchVol.
Alterations in brain extracellular dopamine and glycine levels following combined administration of the glycine transporter type-1 inhibitor Org and risperidone. The therapeutic efficacy of jyranyi atypical antipsychotics is explained in part by antagonism of the dopaminergic neurotransmission, mainly by blockade of D2 dopamine receptors.
Keywords Antipsychotic agents Extracellular glycine and dopamine Glycine transporter type-1 inhibitors Microdialysis Schizophrenia. Risperidone increased extracellular concentrations of dopamine but failed to influence those of glycine or glutamate measured in microdialysis samples. To investigate this type of combined drug administration, rats were treated with zsilt atypical antipsychotic risperidone together with the GlyT-1 inhibitor Org Link to citation list in Scopus.
Interestingly, the extracellular concentrations of glutamate were also enhanced.
Loop | Zsolt Juranyi
Combined drug administration with D2 dopamine receptor jyranyi and activation of hypofunctional NMDA receptors may be needed for a more effective treatment of positive and negative symptoms and the accompanied cognitive deficit in schizophrenia.
N-methyl-d-aspartate NMDA receptor hypofunction in schizophrenia can be reversed by glycine transporter type-1 GlyT-1 inhibitors, which regulate glycine concentrations at the vicinity of NMDA receptors.
T1 – Alterations in brain extracellular dopamine and glycine levels following combined administration of the glycine transporter type-1 inhibitor Org and risperidone. The therapeutic efficacy of all atypical antipsychotics is explained in part by antagonism of the dopaminergic neurotransmission, mainly by blockade of D 2 dopamine receptors.
The most dominant hypotheses for the pathogenesis of schizophrenia have focused primarily upon hyperfunctional dopaminergic and hypofunctional jurayni neurotransmission in the central nervous system.
Access to Document Neurochemical Research35 12 Combined drug administration with D 2 dopamine receptor blockade and activation of hypofunctional NMDA receptors may be needed for a more effective treatment of positive and negative symptoms and the accompanied cognitive deficit in schizophrenia. When risperidone and Org were added in combination, a decrease in extracellular dopamine concentrations was accompanied with sustained elevation of extracellular glycine levels.
zsoolt
Abstract The most dominant hypotheses for the pathogenesis of schizophrenia have focused primarily upon hyperfunctional dopaminergic and hypofunctional glutamatergic neurotransmission in the central nervous system.
